Frequently Asked Questions

Here’s the list of our most frequently asked Riordan Clinic Questions. If you have a question you’d like answered and can’t find it in the list, please feel free to call us: 316-682-3100 or contact us.

Research

  • Do you have any information or research on veterans with Alzheimers or other medical conditions supporting that vitamins have helped or aided?

    We have not conducted research on the effect of vitamins on patients with Alzheimers disease. However, recent studies by researchers at Oxford university have demonstrated that daily doses of vitamin B can reduce by 50% the rate of brain shrinkage in elderly people with mild cognitive impairment, a precursor to Alzheimer’s.

  • My insulin reading device shows a higher level of glucose after intravenous vitamin C. Can ascorbic acid have an effect on increasing the blood glucose level?

    No, high dose intravenous vitamin C (IVC) at levels of 15 grams and higher will cause a false positive on finger-stick blood glucose strips (electrochemical method) read on various glucometers. Depending on the dose, the false positive glucose and occasionally “positive ketone” readings may last for eight hours after the infusion. Blood taken from a vein and run in a laboratory using the hexokinase serum glucose method is not affected! The electrochemical strip cannot distinguish between ascorbic acid and glucose at high levels due to the similarity of the vitamin C molecule and the glucose molecule. Oral vitamin C supplementation does not have this effect.

    Reference: Jackson JA, Hunninghake RE, et al. False positive blood glucose readings after high-dose intravenous vitamin C. J Orthomol Med, 2006; 21(4):188-190.

  • Do you have proof that your C-Statin (from bind weed) has an effect on the suppression of blood vessel formation in diabetic retinopathy?

    The supplement C-Statin, which was developed at Riordan Clinic, was tested for a variety of effects. In those studies, it was demonstrated that one of its primary modes of action was inhibition of angiogenesis (angiogenesis = new blood vessel formation) and a secondary mode of action was enhancement of natural killer cell activity. While this supplement was not tested specifically for prevention or reduction of angiogenesis related to diabetic retinopathy, it is a general angiogenesis inhibitor. Therefore, there is no reason to suspect that it would not inhibit the angiogenesis in diabetic retinopathy. The extract proved to inhibit angiogenesis in a variety of cancer models and various known chemical enhancers of angiogenesis.